Sahil Sharma¹*, Vidula Gujjarwar², Nidhi Kushwaha³, Devaki Krishna³ and Bhavika Sahu³

¹PG Scholar (3^rd Year), Department of Rog Nidan Evum Vikruti Vigyan, Chaudhary Brahm Prakash Ayurved Charak Sansthan, Khera Dabar, New Delhi – 110073, India

²Professor, HOD, Department of Rog Nidan Evum Vikruti Vigyan, Chaudhary Brahm Prakash Ayurved Charak Sansthan, Khera Dabar, New Delhi – 110073, India

³Department of Rog Nidan Evum Vikruti Vigyan, Chaudhary Brahm Prakash Ayurved Charak Sansthan, Khera Dabar, New Delhi – 110073, India

*Corresponding Author: Sahil Sharma, PG Scholar (3 rd Year), Department of Rog Nidan Evum Vikruti Vigyan, Chaudhary Brahm Prakash Ayurved Charak Sansthan, Khera Dabar, New Delhi – 110073, India.

Received: January 20, 2026; Published: February 28, 2026

Abstract

Background: Hypertension is a major global public health challenge and a leading risk factor for cardiovascular, cerebrovascular, and renal morbidity and mortality. Although hypertension is extensively studied in modern medicine, its etiopathogenesis remains largely descriptive. Ayurveda offers a unique interpretative model; however, a standardized explanation of hypertension with reference to specific dietary factors such as Ati Lavaṇa Rasa (~excessive salt intake) is still lacking. This review attempts to bridge this conceptual gap by integrating classical Ayurvedic principles with contemporary scientific understanding.

Objective: * To analyze classical Ayurvedic references related to Lavaṇa Rasa and Rakta-Pitta Dushti, * To correlate Ayurvedic concepts with the modern pathophysiology of hypertension. * To highlight preventive implications through Nidāna Parivarjana (~avoidance of causative factor).

Materials and Methods: A comprehensive review of classical Ayurvedic texts, including Charaka Samhita, Sushruta Samhita, and Ashtanga Hridaya, was undertaken. Relevant contemporary medical literature, published research articles, and recent scientific updates from standard biomedical databases were also analyzed to elucidate the etiological factors and pathophysiological mechanisms of hypertension.

Results: Excessive consumption of Lavaṇa Rasa leads to Pitta and Rakta (~blood) aggravation, resulting in Rakta Ati-Vriddhi, increased blood volume, and elevated cardiac output. Aggravation of Ranjaka Pitta contributes to increased blood viscosity, while Sādhaka Pitta influences cardiac rhythmicity and workload and causes emotional and mental disturbances leading to stress, collectively promoting hypertension. These Ayurvedic mechanisms show close parallels with modern concepts of hypervolemia, increased blood viscosity, stress, cardiac output elevation,

Conclusion: Hypertension can be comprehensively understood as a multifactorial disorder involving the interplay of Vata, Pitta, and Kapha through disturbances in Dosha, Dhatu, and Srotas. Excess salt intake acts as a key modifiable etiological factor. Integrating Ayurvedic principles with modern scientific knowledge may provide a broader conceptual framework for understanding hypertension and may support the development of holistic preventive and therapeutic strategies.

Keywords: Hypertension; Lavaṇa Rasa; Rakta Ati Vridhi; Ayurveda

References

1. World Health Organization. “Global brief on hypertension”. Geneva: World Health Organization (2025).
2. Gupta R. “Trends in hypertension epidemiology in India”. Journal of Human Hypertension 18 (2004): 73-78.
3. Kannel WB. “Hypertension: reflections on risks and prognostication”. Medical Clinics of North America 3 (2008): 541-548.
4. Chen S. “Essential hypertension: perspectives and future directions”. Journal of Hypertension1 (2012): 42-45.
5. Nguyen Q., et al. “Hypertension management: an update”. American Health and Drug Benefits1 (2010): 47-56.
6. Ralston SH., et al. “Davidson’s principles and practice of medicine”. 23^rd Edinburgh: Elsevier. Chapter 16 (2018): 508.
7. Shukla V and Tripathi R. “Charaka Samhita (Vaidyamanorama Hindi commentary)”. Vol. 1. Varanasi: Chaukhamba Sanskrit Pratishthan; 2017. Chikitsa Sthana, Chapter 26, Verse 42.3 (2017): 370.
8. Shastri AD. “Sushruta Samhita (Ayurveda Tattva Sandipika Hindi commentary)”. Vol. 1. Sutra Sthana, Chapter 15, Verse 78. Varanasi: Chaukhamba Sanskrit Sansthan; (2018).
9. Hall JE. “Guyton and Hall textbook of medical physiology”. 13^th Philadelphia: Elsevier. Chapter 19 (2016): 219.
10. Letcher RL., et al. “Direct relationship between blood pressure and blood viscosity in normal and hypertensive subjects”. American Journal of Medicine6 (1981): 1195-1202.
11. Sembulingam K and Sembulingam P. “Properties of cardiac muscle”. In: Essentials of medical physiology. 5^th New Delhi: Jaypee Brothers Medical Publishers; (2010): 509.
12. Tripathi B. “Ashtanga Hridaya (Nirmala Hindi commentary)”. Sutra Sthana, Chapter 1, Verse 11. New Delhi: Chaukhamba Sanskrit Pratishthan; (2019): 10.
13. Tripathi B. “Ashtanga Hridaya (Nirmala Hindi commentary)”. Sutra Sthana, Chapter 12, Verse 13. New Delhi: Chaukhamba Sanskrit Pratishthan; (2019): 172.
14. Kulkarni S., et al. “Stress and hypertension”. WMJ 11 (1998): 34-38.

Citation

Citation: Sahil Sharma., et al. “Role of Ati Lavana Rasa in the Etiopathogenesis of Hypertension: An Integrative Ayurvedic Perspective". Acta Scientific Nutritional Health 10.3 (2026): 34-38. Evaluation of Probiotic Akkermansia muciniphila Strain in Patients with Irritable Bowel Syndrome". Acta Scientific Nutritional Health 10.3 (2026): 23-33.

Copyright

Copyright: © 2026 Sahil Sharma., et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.